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To prevent the development of this pathology, people must take precautions: It is necessary to destroy kissing bugs, which are carriers of parasites. Rooms in which insects were seen should be treated with insecticides. If a person is to have a blood transfusion, then he must be convinced of the quality of the biological material.
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    Causes of adalat disease Symptoms of Chagas disease Diagnosis of Chagas disease Treatment and prognosis of Chagas disease Prices for treatment.

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    Chagas disease (American trypanosomiasis) is a protozoosis caused by an intracellular protozoan parasite of the genus Trypanosoma (T. cruzi), leading to inflammatory and degenerative changes in the heart, nervous system, and gastrointestinal tract.

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    Chagas disease refers to natural focal diseases, is widespread in almost all countries of nifedipine continent: Central and South America are endemic for trypanosomiasis.

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    In rural areas, the majority of the population becomes infected with trypanosomiasis in childhood, often the invasion is asymptomatic. Among those infected with Chagas disease, males predominate.

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  • Infection of bedbugs occurs in the process of feeding on human or animal blood containing trypomastigotes. The causative agent of Chagas disease is introduced into the human body along with infected feces of bedbugs when combing wounds after their bites on the skin and mucous membranes of the lips, nose, and conjunctiva. A characteristic feature of T. cruzi is the ability to intracellular parasitism in macrophages of the skin and mucous membranes, in myocardial cells, endothelial cells of lymph nodes, spleen, liver, lungs, and neuroglia.

  • After the rupture of the affected cells, overflowing with multiplying amastigotes, infection of new nifedipine occurs. Parasitization of trypanosomes leads to inflammatory-dystrophic and degenerative damage to internal organs. The most common way of infection with Chagas disease is transmissible, transmission is possible by alimentary, sexual, transplacental, blood transfusion and organ transplantation. T. cruzi is characterized by persistence in the host organism throughout life.

  • The incubation period of the disease lasts 1-3 weeks. Then, at the site of penetration of trypanosomes, a local inflammatory reaction (step) may develop in the form of an erythematous node with swelling and redness, or unilateral purple edema of the eyelid (Romigny's symptom) with conjunctivitis, accompanied by an increase in regional lymph nodes. Chagas disease passes in 2 stages: acute (the first 2 months) with the circulation of a large number of parasites in the blood and chronic - with a concentration of trypanosomes in the internal organs.

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    In the chronic form of Chagas disease, there may be no symptoms for a long time until irreversible damage to the internal organs occurs. Most often, cardiomyopathy develops, which is expressed in heart failure, arrhythmia, thromboembolism. The gastrointestinal tract is characterized by pathological expansion of the esophagus (megaesophagus), manifested by dysphagia, pain when swallowing, and expansion of the large intestine (megacolon), accompanied by intestinal obstruction, accumulation of fecal stones.

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    There are autonomic disorders and peripheral neuropathy. Over time, Chagas disease can lead tosudden death due to progressive destruction of the heart muscle. Diagnosis of Chagas disease includes a thorough history taking (taking into account the patient's place of birth, trips to areas endemic for the disease), analysis of clinical data and laboratory studies (microscopic and cultural methods, serological tests and PCR).

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    Bacteriological culture of the patient's blood helps to identify a pure culture of T. cruzi. In Chagas disease, xenodiagnostics is used, including the study of the contents of the intestine of a non-invasive triatomic bug after it has been saturated with the patient's blood. It is possible to conduct a biological test with the introduction of the patient's blood to guinea pigs or white mice and the subsequent study of tissue samples.

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